Please use this identifier to cite or link to this item: http://223.31.159.10:8080/jspui/handle/123456789/1305
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dc.contributor.authorSingh, Yeshveer-
dc.contributor.authorVerma, Praveen K.-
dc.date.accessioned2022-03-15T07:10:10Z-
dc.date.available2022-03-15T07:10:10Z-
dc.date.issued2022-
dc.identifier.citationTrends in Plant Science, 27(6): 513-515en_US
dc.identifier.issn1360-1385-
dc.identifier.otherhttps://doi.org/10.1016/j.tplants.2022.02.007-
dc.identifier.urihttps://www.sciencedirect.com/science/article/pii/S1360138522000498-
dc.identifier.urihttp://223.31.159.10:8080/jspui/handle/123456789/1305-
dc.descriptionAccepted date: 9 March 2022en_US
dc.description.abstractStomata offer an effortless opportunity for pathogens to enter host plants and exploit that resource. Upon pathogen attack, stomatal closure is a commonly observed response to prevent microbial invasion. A recent study by Zou et al. shows that stomatal closure following exposure to microbe-associated molecular patterns (MAMPs) is mediated by altered actin dynamics in an MPK3/6 phosphorylation- and VLN3-dependent manner.en_US
dc.description.sponsorshipY.S. thanks the NIPGR and Department of Biotechnology (DBT), Government of India, for research fellowships.en_US
dc.language.isoen_USen_US
dc.publisherElsevier B.V.en_US
dc.subjectMPK3/6–VLN3en_US
dc.subjectstomatal defenseen_US
dc.titleGuiding the guards: MPK3/6-VLN3 module regulating stomatal defenseen_US
dc.typeArticleen_US
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